What did @ivanmartellato actually say?
Honestly, the transcript here is nearly incoherent. The audio-to-text capture produced fragments about "schizophrenia of my life" and "BTC-157" with no legible nutritional argument. What we can work with is the caption, which frames butter as "an ally for gut health" and name-drops butyric acid, lactose, curcumin, berberine, quercetin, ginger, and BPC-157 as related concepts. The creator appears to be building a case that butter, specifically its short-chain fatty acid content, benefits intestinal health. The hashtag structure suggests a broader anti-inflammatory supplement framing. We can't quote the video directly because the transcript is garbled, but the caption argument is clear enough to fact-check on its own merits.
Does the science back this up?
Partially, yes, but the framing oversimplifies the evidence considerably. Butter does contain butyrate, but in amounts that won't meaningfully shift colonic butyrate concentrations compared to fermentation-derived butyrate from dietary fiber.
Butyrate is a short-chain fatty acid that serves as the primary energy source for colonocytes, the cells lining your colon. This part is well-established. Canani et al. (2011, Molecules) reviewed butyrate's role in intestinal barrier function and found genuine anti-inflammatory effects in mucosal tissue. However, most dietary butyrate from butter is absorbed in the small intestine before it reaches the colon. The colon's butyrate primarily comes from microbial fermentation of resistant starch and soluble fiber, not from eating butter directly. Louis and Flint (2009, FEMS Microbiology Letters) documented this clearly. So butter as a direct butyrate delivery vehicle for the gut? Weak mechanism.
What did they get wrong (or right)?
They got the general concept directionally right but mechanistically sloppy. Butyrate matters for gut health. That is not controversial. Where this goes sideways is the implication that eating butter is a meaningful strategy for boosting colonic butyrate levels.
A 100g serving of butter contains roughly 3-4g of butyric acid. But absorption kinetics mean very little of this survives to the colon intact. By contrast, consuming 20-30g of fermentable fiber daily can generate far more butyrate in situ through microbial fermentation. Associating butter consumption with gut repair without that context is selective at best. The BPC-157 hashtag is also worth flagging. BPC-157 is a synthetic peptide, not a food compound, and bundling it casually with dietary claims creates a misleading equivalence. BPC-157 has shown gut mucosal healing effects in rodent models (Sikiric et al., 2016, Current Pharmaceutical Design), but human clinical data is limited and it is not an approved therapeutic in most jurisdictions. Dropping it as a hashtag without that distinction is irresponsible shorthand.
What should you actually know?
If gut health is the actual goal, the butyrate conversation is worth having, just more carefully than this video frames it. Fermented foods, resistant starch, and high-fiber diets are the evidence-backed routes to raising colonic butyrate. Butter is not harmful for most people, and it is not nothing, but it is a poor primary strategy for gut microbiome support.
For the anti-inflammatory compounds listed in the hashtags: curcumin has absorption problems that limit bioavailability without piperine or lipid-based delivery (Anand et al., 2007, Molecular Pharmaceutics). Berberine has more convincing gut-related data, including effects on the microbiome and intestinal permeability (Feng et al., 2018, Frontiers in Pharmacology). Quercetin is promising but human data is inconsistent. These are not substitutes for each other, and stacking them without understanding interactions or doses is not something any responsible source should encourage. If you are considering peptides like BPC-157 for gut issues, that conversation needs to happen with a licensed clinician, not an Instagram caption.