What did @zack.chug actually say?
In a 78K-view TikTok, @zack.chug describes noticing signs of low testosterone, including "more gyno, low energy, low libido and brain fog," and blames chronic stress and cortisol for suppressing his T levels. He then outlines a self-prescribed protocol: an extra hour of sleep, warm milk with turmeric before bed, daily vitamin D and magnesium, more dietary fat from avocados, eggs, and butter, and heavy compound lifting. The framing is confident and specific, which is exactly when you need to slow down and check the receipts.
To his credit, none of this is selling a product or pushing a dangerous stack. These are lifestyle interventions, and some of them are grounded in real physiology. But the way he connects each dot, and a few of the dots themselves, deserves a closer look.
Does the science back this up?
Mostly, yes, with some meaningful caveats. The cortisol-testosterone connection is real and well-documented. Sleep's role in testosterone production is also solid science. The dietary fat claim has legitimate support. The vitamin D and magnesium evidence is real but weaker than the confident delivery implies.
The HPG axis claim is accurate in broad strokes: the hypothalamic-pituitary-gonadal axis is responsible for regulating testosterone, and sleep, particularly slow-wave sleep, is when the bulk of the luteinizing hormone pulses that drive testosterone production occur. Leproult and Van Cauter (2011, JAMA) showed that just one week of sleep restricted to five hours per night reduced testosterone levels by 10 to 15 percent in young healthy men. That is not trivial.
On dietary fat: a 1984 study by Hamalainen et al. in Hormone and Metabolic Research found that a low-fat diet reduced serum testosterone in men. Cholesterol is genuinely the precursor to steroid hormones, so the "building block" framing is chemically accurate. The compound lifting claim also holds up: resistance training acutely raises testosterone, though the chronic effect on baseline levels in healthy men is more modest (Kraemer and Ratamess, 2005, Sports Medicine).
What did they get wrong (or right)?
The warm milk and turmeric claim is where things get shaky. @zack.chug says warm milk "contains tryptophan which boosts melatonin for deeper sleep." Milk does contain tryptophan, but the amount is small enough that its direct effect on melatonin synthesis via a glass of warm milk is not well-supported in healthy adults. A 2022 review in Nutrients by Sutanto et al. found that tryptophan supplementation studies use doses far above what a glass of milk provides. The effect, if any, is likely modest and indirect.
The turmeric claim is essentially decoration. Curcumin has shown some anti-inflammatory properties in concentrated supplement form, but there is no credible evidence that turmeric in warm milk meaningfully raises melatonin or improves testosterone. This is a case of stacking plausible-sounding ingredients without the evidence to connect them.
The gynecomastia mention is worth flagging too. "More gyno" is a clinical sign, not just a lifestyle symptom. Gynecomastia can signal an unfavorable estrogen-to-testosterone ratio, liver issues, or medication effects. Recommending sleep and avocados in response to gynecomastia is not a substitute for getting bloodwork done.
What he got right: the stress-cortisol-testosterone loop is real. The sleep recommendation is well-supported. Dietary fat and cholesterol as hormone precursors is legitimate biochemistry. Heavy compound lifting as a tool for hormonal health is supported by evidence.
What should you actually know?
If you are experiencing symptoms like low libido, fatigue, brain fog, and changes in body composition, lifestyle adjustments like better sleep, resistance training, and adequate dietary fat are genuinely reasonable starting points. But they are not a diagnosis, and they are not a treatment plan for actual hypogonadism.
Clinically low testosterone is defined by bloodwork, not by how you feel after a stressful week. The Endocrine Society defines hypogonadism as total testosterone below roughly 300 ng/dL, confirmed on at least two morning samples. Symptoms alone are not enough to diagnose it, and lifestyle changes alone are not always enough to fix it.
Vitamin D deficiency is common, and correcting it has shown modest benefits for testosterone in deficient men specifically. Pilz et al. (2011, Hormone and Metabolic Research) found that vitamin D supplementation raised testosterone in deficient men by about 25 percent. But if you are not deficient, the effect is likely negligible. Magnesium has similar nuance: it matters when you are low, but it is not a universal testosterone booster.
The bottom line is that @zack.chug's protocol is not dangerous, and parts of it are genuinely useful. But the confident, mechanistic framing, "this activates your HPG axis," glosses over the fact that these are population-level associations, not guaranteed individual outcomes. If your symptoms are persistent, get your labs done.