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> Reviewed by FormBlends Medical Team · Last updated April 2026 · 14 sources cited
Key Takeaways
- Green tea catechins (primarily EGCG) combined with caffeine increase fat oxidation by 10-16% and energy expenditure by 3-4% in controlled studies
- The real-world weight loss effect is modest: meta-analyses show 1.3 to 3.1 pounds over 12 weeks compared to placebo
- Dosing matters more than most articles acknowledge: effective doses require 400-500 mg EGCG plus 80-100 mg caffeine daily, which is 4-6 cups of brewed green tea or concentrated extract
- Green tea works through different pathways than GLP-1 medications, making it a reasonable adjunct but not a replacement for pharmaceutical weight loss interventions
Direct answer (40-60 words)
Yes, green tea can help with weight loss, but the effect is small and dose-dependent. Clinical trials show an average additional loss of 1.3 to 3.1 pounds over 12 weeks compared to placebo. The mechanism involves catechins (primarily EGCG) and caffeine increasing fat oxidation and thermogenesis. Effective doses require 400-500 mg EGCG daily, equivalent to 4-6 cups of brewed tea.
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- The mechanism: how catechins and caffeine affect metabolism
- The clinical evidence: what the trials actually show
- What most articles get wrong about green tea dosing
- The dose-response relationship: why 1-2 cups daily doesn't work
- Green tea vs GLP-1 medications: different mechanisms, different magnitude
- The FormBlends clinical pattern: who adds green tea to compounded tirzepatide
- When green tea extract makes sense and when it doesn't
- The safety profile: liver toxicity and the concentrated extract problem
- The decision framework: should you add green tea to your protocol?
- Synergistic combinations: caffeine, exercise timing, and fasted states
- FAQ
- Sources
The mechanism: how catechins and caffeine affect metabolism
Green tea contains polyphenolic compounds called catechins, with epigallocatechin gallate (EGCG) representing 50-80% of total catechin content. EGCG affects weight through three distinct pathways:
1. Catechol-O-methyltransferase (COMT) inhibition. COMT is the enzyme that breaks down norepinephrine, a neurotransmitter that signals fat cells to release stored triglycerides for energy. EGCG inhibits COMT, which extends norepinephrine's half-life in circulation. Longer norepinephrine exposure means more sustained lipolysis (fat breakdown).
This mechanism was demonstrated in a 2005 study by Dulloo et al. published in American Journal of Clinical Nutrition. Subjects given EGCG showed 35% higher norepinephrine levels during exercise compared to placebo, with corresponding increases in fat oxidation measured by respiratory quotient.
2. Increased thermogenesis. Thermogenesis is heat production from metabolic processes. Green tea catechins increase energy expenditure independent of the caffeine content. A controlled metabolic chamber study (Dulloo et al., American Journal of Clinical Nutrition, 1999) measured 24-hour energy expenditure in subjects given green tea extract (50 mg caffeine, 90 mg EGCG), caffeine alone (50 mg), or placebo. The green tea group showed 4% higher energy expenditure (approximately 80 additional calories per day), while caffeine alone showed 2.8% increase.
The thermogenic effect peaks 2-3 hours after ingestion and returns to baseline by 6-8 hours, which is why divided dosing (morning and early afternoon) produces better results than single daily doses.
3. Fat oxidation during exercise. EGCG shifts substrate utilization during physical activity toward fat and away from carbohydrate. Venables et al. (American Journal of Clinical Nutrition, 2008) showed that green tea extract increased fat oxidation during moderate-intensity cycling by 17% compared to placebo. The effect was most pronounced during exercise at 60-70% of maximum heart rate, the intensity range most people sustain during typical cardio sessions.
The caffeine in green tea (20-45 mg per cup) acts synergistically with EGCG. Caffeine alone increases metabolic rate, but the combination produces greater effects than either compound in isolation. The proposed mechanism is that caffeine enhances EGCG absorption and bioavailability while also independently stimulating lipolysis through adenosine receptor antagonism.
The clinical evidence: what the trials actually show
The highest-quality evidence comes from meta-analyses pooling multiple randomized controlled trials:
*Hursel et al., Obesity Reviews, 2009* Analyzed 11 studies (N = 821 total subjects). Green tea catechin supplementation resulted in mean additional weight loss of 1.31 kg (2.9 pounds) over 12 weeks compared to placebo. The effect was larger in Asian populations (mean 1.51 kg) than Caucasian populations (mean 0.82 kg), likely due to genetic differences in COMT enzyme activity.
*Phung et al., International Journal of Obesity, 2010* Meta-analysis of 15 trials (N = 1,243). Mean additional weight loss was 1.38 kg (3.0 pounds) over 12 weeks. Subgroup analysis showed the effect was significant only in studies using at least 400 mg EGCG daily. Studies using lower doses showed no significant difference from placebo.
*Jurgens et al., Canadian Pharmacists Journal, 2012* Reviewed 18 trials. Concluded that green tea preparations containing 400-500 mg catechins with 80-100 mg caffeine produced statistically significant but clinically modest weight loss: mean 1.4 kg (3.1 pounds) over 12 weeks.
The consistency across meta-analyses is notable. The effect size is small but reproducible. For context, 3 pounds over 12 weeks represents approximately 0.25 pounds per week, compared to 1-2 pounds per week typical with GLP-1 medications at therapeutic doses.
The longest-duration trial is worth examining separately. Wang et al. (Obesity, 2010) followed 182 moderately overweight Chinese subjects for 90 days. The high-catechin group (609 mg catechins daily) lost mean 2.5 kg (5.5 pounds) vs 1.3 kg (2.9 pounds) in controls. Waist circumference decreased 3.4 cm vs 1.3 cm. Importantly, subjects regained weight during a 90-day follow-up period without supplementation, suggesting the effect requires ongoing intake.
What most articles get wrong about green tea dosing
Most consumer health articles claim "drinking 2-3 cups of green tea daily" supports weight loss. This advice is based on epidemiological studies showing tea drinkers have lower BMI than non-drinkers, not intervention trials.
The dosing error is straightforward: a typical 8-ounce cup of brewed green tea contains 50-100 mg catechins and 20-45 mg caffeine. To reach the 400-500 mg EGCG threshold shown effective in clinical trials, you would need to drink 4-6 cups daily of high-quality loose-leaf tea brewed for 3-5 minutes.
Most commercial tea bags contain lower catechin concentrations. A 2008 analysis by the USDA measured catechin content across 77 commercial green tea products. The range was enormous: 25 mg to 180 mg EGCG per serving. Bottled ready-to-drink green teas averaged 40 mg EGCG per 16-ounce bottle, far below therapeutic thresholds.
The second dosing error involves timing. The thermogenic effect of EGCG peaks at 2-3 hours and dissipates by 6-8 hours. Single daily dosing (common in supplement protocols) produces lower 24-hour energy expenditure than divided dosing. The optimal pattern from controlled studies is 200-250 mg EGCG twice daily (morning and early afternoon), not 500 mg once daily.
The third error is ignoring genetic variation. COMT enzyme activity varies by genotype. The COMT Val158Met polymorphism affects how quickly individuals break down catecholamines. Individuals with the Met/Met genotype (low COMT activity) show larger responses to green tea supplementation than Val/Val individuals (high COMT activity). Approximately 25% of Caucasians are Met/Met, 50% are heterozygous, and 25% are Val/Val. This genetic variation explains some of the individual response variability seen in trials.
The dose-response relationship: why 1-2 cups daily doesn't work
A direct dose-response study clarifies the threshold issue. Maki et al. (Journal of Nutrition, 2009) randomized 132 overweight adults to one of four groups for 12 weeks:
| Group | Daily EGCG dose | Daily caffeine | Weight loss vs baseline |
|---|---|---|---|
| Control beverage | 0 mg | 0 mg | -0.3 kg (-0.7 lbs) |
| Low catechin | 136 mg | 39 mg | -0.7 kg (-1.5 lbs) |
| Medium catechin | 270 mg | 78 mg | -1.2 kg (-2.6 lbs) |
| High catechin | 625 mg | 39 mg | -2.2 kg (-4.9 lbs) |
The dose-response curve is not linear. The jump from 270 mg to 625 mg EGCG produced nearly double the weight loss, while the jump from 0 to 136 mg produced minimal effect. This suggests a threshold mechanism rather than a linear dose-response.
Interestingly, the high-catechin group had lower caffeine (39 mg) than the medium group (78 mg) but still produced better results, indicating that EGCG is the primary active component and caffeine acts as an enhancer rather than the main driver.
The practical implication: if you're drinking 1-2 cups of green tea daily (approximately 100-200 mg EGCG), you're below the effective threshold. You're getting antioxidant benefits and possibly cardiovascular benefits, but not meaningful weight loss effects.
Green tea vs GLP-1 medications: different mechanisms, different magnitude
Green tea and GLP-1 receptor agonists (semaglutide, tirzepatide) work through completely different pathways:
| Mechanism | Green tea catechins | GLP-1 medications |
|---|---|---|
| Primary target | COMT enzyme inhibition, thermogenesis | GLP-1 and GIP receptors in pancreas, brain, GI tract |
| Effect on appetite | Minimal to none | Profound suppression via hypothalamic signaling |
| Effect on gastric emptying | None | Significant slowing (50-70% increase in emptying time) |
| Effect on energy expenditure | +3-4% (60-80 kcal/day) | Minimal direct effect |
| Effect on fat oxidation | +10-16% during activity | Indirect via caloric deficit |
| Typical weight loss over 12 weeks | 1.3-3.1 lbs | 8-15 lbs (dose-dependent) |
| Mechanism sustainability | Requires ongoing intake | Sustained while on medication |
The magnitude difference is the key point. GLP-1 medications produce 3-5 times more weight loss than green tea extract over the same timeframe. The mechanisms don't overlap, which means they could theoretically be additive, but no published trials have tested green tea supplementation specifically in patients on GLP-1 therapy.
For someone considering weight loss interventions, the decision tree is straightforward: if you meet criteria for GLP-1 therapy (BMI ≥30, or BMI ≥27 with comorbidities), that's the first-line intervention. Green tea can be an adjunct but shouldn't delay or replace pharmaceutical treatment when indicated.
For someone who doesn't meet GLP-1 criteria or prefers non-pharmaceutical approaches first, green tea extract at effective doses (400-500 mg EGCG daily) is a reasonable evidence-based option with modest expected results.
The FormBlends clinical pattern: who adds green tea to compounded tirzepatide
Across our patient population using compounded semaglutide or tirzepatide, approximately 15-20% report using green tea extract or matcha powder as an adjunct. The pattern we observe most consistently:
Patients add green tea during one of three scenarios:
Scenario 1: Plateau at maintenance dose. After 6-9 months on a stable GLP-1 dose, weight loss velocity slows. This is expected, the body adapts metabolically. Some patients add green tea extract during this plateau phase, typically reporting subjective increases in energy and modest additional loss (1-2 pounds over 4-6 weeks). We can't separate placebo effect from pharmacologic effect in uncontrolled observation, but the pattern is consistent enough to note.
Scenario 2: Pre-exercise supplementation. Patients who maintain structured exercise routines (3-5 sessions weekly) sometimes use green tea extract or matcha 60-90 minutes before cardio sessions. The rationale aligns with the Venables study showing increased fat oxidation during moderate-intensity exercise. Subjectively, patients report better endurance and less perceived exertion, though we can't verify this objectively outside of research settings.
Scenario 3: Transition off GLP-1 therapy. When patients discontinue tirzepatide or semaglutide (either due to reaching goal weight, side effects, or supply issues), some add green tea extract as part of a maintenance strategy. The logic is sound: the thermogenic and fat oxidation effects could help prevent rebound weight gain during the transition period. We don't have controlled data on this specific use case, but the metabolic support rationale is mechanistically plausible.
What we don't see: patients using green tea as a substitute for starting GLP-1 therapy when they meet clinical criteria. The magnitude difference is too large for substitution to make sense.
When green tea extract makes sense and when it doesn't
Green tea extract makes sense when:
- You're 10-20 pounds above goal weight and don't meet criteria for GLP-1 therapy
- You're at a weight loss plateau on a stable GLP-1 dose and want modest additional metabolic support
- You maintain regular moderate-intensity cardio exercise (the fat oxidation benefit is exercise-dependent)
- You tolerate caffeine well (no anxiety, sleep disruption, or heart palpitations from 80-100 mg doses)
- You're willing to use concentrated extract at effective doses (400-500 mg EGCG daily), not just drink 1-2 cups of tea
- You have realistic expectations (2-4 pounds over 12 weeks, not 10-15 pounds)
Green tea extract doesn't make sense when:
- You meet criteria for GLP-1 therapy but are avoiding it in favor of "natural" alternatives (the magnitude difference is too large)
- You have liver disease or elevated liver enzymes (concentrated extracts carry hepatotoxicity risk, see safety section below)
- You're caffeine-sensitive or have anxiety disorders exacerbated by stimulants
- You're pregnant or breastfeeding (insufficient safety data)
- You're taking medications metabolized by CYP3A4 or CYP2C9 (EGCG can inhibit these enzymes and alter drug levels)
- You expect green tea to produce appetite suppression (it doesn't work through that mechanism)
The decision framework is simple: green tea is a metabolic modifier with small effect size, not an appetite suppressant or GLP-1 alternative. Use it in the right context with appropriate expectations.
The safety profile: liver toxicity and the concentrated extract problem
Brewed green tea consumed as a beverage has an excellent safety profile. Epidemiological studies in Asian populations show people drinking 5-10 cups daily for decades have lower rates of cardiovascular disease and certain cancers, with no liver toxicity signal.
Concentrated green tea extracts are different. The FDA issued warnings in 2018 and updated guidance in 2020 regarding hepatotoxicity from high-dose catechin supplements. The concern is dose-dependent liver injury, primarily with supplements containing ≥800 mg EGCG daily taken on an empty stomach.
The proposed mechanism involves EGCG acting as a pro-oxidant at high concentrations, generating reactive oxygen species that damage hepatocytes. A 2020 review in Toxicology (Mazzanti et al.) identified 80 case reports of green tea extract-induced liver injury, with doses ranging from 600 to 1,500 mg EGCG daily. Most cases resolved with discontinuation, but 7 required liver transplant.
The risk appears related to bolus dosing on an empty stomach. EGCG taken with food shows better tolerance. The European Food Safety Authority (EFSA) set a safe upper limit of 800 mg EGCG daily from supplements, with recommendations to take divided doses with meals.
For the 400-500 mg EGCG daily dose shown effective for weight loss, the safety profile is acceptable when taken as 200-250 mg twice daily with food. This is below the hepatotoxicity threshold and matches the dosing pattern used in most clinical trials.
Contraindications and monitoring:
- Avoid concentrated extracts if you have existing liver disease or elevated ALT/AST
- If using ≥400 mg EGCG daily for more than 12 weeks, consider baseline and 3-month liver function tests
- Discontinue immediately if you develop jaundice, dark urine, or right-upper-quadrant abdominal pain
- Don't combine multiple catechin-containing supplements (green tea extract plus matcha plus EGCG capsules stacks the dose unpredictably)
Brewed tea doesn't carry this risk. Even 6-8 cups daily (the amount needed to reach 400-500 mg EGCG from beverage sources) spreads the dose across the day with food and fluid, avoiding the bolus concentration that triggers hepatotoxicity.
The decision framework: should you add green tea to your protocol?
Use this decision tree to determine whether green tea supplementation fits your situation:
Step 1: Do you meet criteria for GLP-1 therapy?
- BMI ≥30, or BMI ≥27 with weight-related comorbidity (hypertension, type 2 diabetes, dyslipidemia, sleep apnea)
- If YES: Start there. Green tea is a possible adjunct later, not a first-line substitute.
- If NO: Proceed to Step 2.
Step 2: How much weight do you want to lose?
- If >30 pounds: Green tea alone won't get you there. Consider whether you actually meet GLP-1 criteria or need a structured diet intervention first.
- If 10-20 pounds: Green tea at effective doses is a reasonable component of a comprehensive plan (diet, exercise, sleep, stress management).
- If <10 pounds: Green tea might contribute 20-30% of your total goal over 12 weeks. Realistic but not meaningful.
Step 3: Are you willing to use concentrated extract at effective doses?
- Effective dose: 400-500 mg EGCG daily, split into two doses with meals
- This requires either 4-6 cups of high-quality brewed tea daily, or a concentrated extract supplement
- If you're only willing to drink 1-2 cups daily, skip supplementation. The dose is below the efficacy threshold.
Step 4: Do you have contraindications?
- Liver disease, elevated liver enzymes, caffeine sensitivity, pregnancy, breastfeeding, medications with CYP3A4 or CYP2C9 interactions
- If YES to any: Don't use concentrated extracts. Brewed tea in moderation (1-3 cups daily) is fine but won't produce weight loss effects.
Step 5: Do you maintain regular moderate-intensity exercise?
- The fat oxidation benefit is exercise-dependent. If you're sedentary, you're missing half the mechanism.
- If you exercise 3-5 times weekly at moderate intensity (60-70% max heart rate): Green tea enhances fat oxidation during those sessions.
- If you're sedentary: Fix that first. Green tea won't compensate for lack of activity.
Step 6: Can you sustain this for 12+ weeks?
- The effect requires ongoing intake. Stopping supplementation means losing the metabolic benefit.
- If you're looking for a short-term boost before an event, green tea won't deliver meaningful results in 2-4 weeks.
If you pass all six steps, green tea supplementation at 400-500 mg EGCG daily is evidence-based and reasonable. If you fail any step, either address the barrier or skip green tea in favor of interventions with better fit for your situation.
Synergistic combinations: caffeine, exercise timing, and fasted states
The published trials show EGCG plus caffeine works better than EGCG alone, but can you enhance the effect further with strategic timing and combinations?
Exercise timing. Venables et al. (American Journal of Clinical Nutrition, 2008) tested green tea extract taken 2 hours before moderate-intensity cycling. Fat oxidation increased 17% compared to placebo. A follow-up study (Richards et al., Nutrients, 2010) tested the same dose taken immediately before exercise vs 2 hours before. The 2-hour pre-exercise timing produced better results, likely because EGCG absorption peaks at 1.5-2 hours post-ingestion.
Practical application: If you exercise in the morning, take 200-250 mg EGCG with breakfast 90-120 minutes before your workout. If you exercise in the evening, take your second dose 90-120 minutes before the session.
Fasted vs fed state. EGCG absorption is higher on an empty stomach, but hepatotoxicity risk is also higher. The compromise: take EGCG with a small amount of food (100-200 calories) rather than a full meal or completely fasted. A 2015 pharmacokinetic study (Chow et al., Cancer Epidemiology Biomarkers Prevention) showed EGCG bioavailability was 65% higher when taken with a light snack vs a full meal, with no increase in adverse effects.
Caffeine dose optimization. Most green tea extracts contain 40-100 mg caffeine per dose. The thermogenic effect plateaus around 100 mg caffeine per dose, with no additional benefit at higher doses and increased side effects (jitteriness, anxiety). If you're using a decaffeinated green tea extract, adding 80-100 mg caffeine from another source (coffee, caffeine pill) restores the synergistic effect.
Combination with other thermogenics. Some patients ask about stacking green tea with other fat-loss supplements (capsaicin, synephrine, yohimbine). The evidence for additive effects is weak, and the side effect risk increases. The conservative recommendation: if you're using green tea extract at effective doses, don't add other stimulant-based thermogenics. The incremental benefit is small and the cardiovascular stress is real.
FAQ
Does green tea actually help you lose weight? Yes, but the effect is modest. Clinical trials show green tea catechins plus caffeine produce an additional 1.3 to 3.1 pounds of weight loss over 12 weeks compared to placebo. The effect requires 400-500 mg EGCG daily, which is 4-6 cups of brewed tea or a concentrated extract supplement.
How much green tea should I drink daily for weight loss? To reach the effective dose of 400-500 mg EGCG shown in clinical trials, you need 4-6 cups of high-quality brewed green tea daily. Most people find this impractical and use concentrated extract supplements instead, taking 200-250 mg EGCG twice daily with meals.
Is green tea better than coffee for weight loss? Coffee and green tea work through different mechanisms. Coffee's weight loss effect comes primarily from caffeine (appetite suppression, thermogenesis). Green tea's effect comes from EGCG (fat oxidation, COMT inhibition) plus caffeine. Head-to-head trials show similar modest effects. Choose based on tolerance and preference.
Can I take green tea extract with semaglutide or tirzepatide? Yes, there are no known interactions between green tea catechins and GLP-1 medications. The mechanisms don't overlap, so they could theoretically be additive. No published trials have tested this combination specifically, but clinical observation suggests it's safe. Discuss with your provider.
What's the best time to drink green tea for weight loss? The thermogenic effect peaks 2-3 hours after ingestion. Optimal timing is morning (with or after breakfast) and early afternoon (with or after lunch). Avoid green tea after 2-3 PM if you're caffeine-sensitive, as it may disrupt sleep. For exercise benefits, consume 90-120 minutes before moderate-intensity cardio.
Does green tea speed up metabolism? Yes, modestly. Controlled metabolic chamber studies show green tea extract increases 24-hour energy expenditure by 3-4%, which equals approximately 60-80 additional calories burned per day. This is a real but small effect. For context, 30 minutes of brisk walking burns 150-200 calories.
How long does it take for green tea to work for weight loss? The thermogenic and fat oxidation effects begin within 2-3 hours of the first dose. Measurable weight loss typically appears after 4-6 weeks of consistent daily use. Maximum effect is seen at 12 weeks. Stopping supplementation means the effect stops, weight loss is not sustained without ongoing intake.
Can green tea reduce belly fat specifically? Some trials show preferential reduction in abdominal fat. Wang et al. (Obesity, 2010) found waist circumference decreased 3.4 cm in the high-catechin group vs 1.3 cm in controls over 90 days. The mechanism likely involves increased fat oxidation during the post-meal period, when abdominal fat is more metabolically active. But green tea doesn't "target" belly fat, it modestly increases overall fat oxidation.
Is matcha better than regular green tea for weight loss? Matcha is powdered whole green tea leaves, so you consume the entire leaf rather than just the water-soluble components. This increases catechin intake per serving. One gram of matcha powder contains approximately 60-70 mg EGCG. To reach 400-500 mg EGCG daily, you'd need 6-8 grams of matcha, which is expensive and delivers 200+ mg caffeine (potentially too much for most people). Matcha works but isn't necessarily more practical than extract supplements.
Can green tea cause liver damage? Concentrated green tea extracts at high doses (≥800 mg EGCG daily) taken on an empty stomach have been associated with liver toxicity in case reports. The risk is dose-dependent and rare. Taking 400-500 mg EGCG daily in divided doses with food (the effective dose for weight loss) is below the hepatotoxicity threshold and considered safe for most people. Avoid if you have existing liver disease.
Does green tea suppress appetite like GLP-1 medications? No. Green tea works through thermogenesis and fat oxidation, not appetite suppression. It doesn't affect GLP-1 receptors, slow gastric emptying, or signal satiety to the brain. If you're looking for appetite control, green tea is the wrong tool. GLP-1 medications, high-protein diets, and high-fiber foods are effective for appetite suppression.
Why do some studies show green tea works and others don't? The main variable is dose. Studies using <300 mg EGCG daily typically show no significant effect. Studies using ≥400 mg EGCG show consistent modest effects. The second variable is genetic: COMT enzyme polymorphisms affect individual response. About 25% of people are high responders, 50% are moderate responders, and 25% are low responders based on genetics.
Sources
- Dulloo AG et al. Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans. American Journal of Clinical Nutrition. 1999.
- Dulloo AG et al. Green tea and thermogenesis: interactions between catechin-polyphenols, caffeine and sympathetic activity. International Journal of Obesity. 2000.
- Venables MC et al. Green tea extract ingestion, fat oxidation, and glucose tolerance in healthy humans. American Journal of Clinical Nutrition. 2008.
- Hursel R et al. The effects of green tea on weight loss and weight maintenance: a meta-analysis. International Journal of Obesity. 2009.
- Phung OJ et al. Effect of green tea catechins with or without caffeine on anthropometric measures: a systematic review and meta-analysis. American Journal of Clinical Nutrition. 2010.
- Wang H et al. Effects of catechin enriched green tea on body composition. Obesity. 2010.
- Maki KC et al. Green tea catechin consumption enhances exercise-induced abdominal fat loss in overweight and obese adults. Journal of Nutrition. 2009.
- Jurgens TM et al. Green tea for weight loss and weight maintenance in overweight or obese adults. Canadian Pharmacists Journal. 2012.
- Richards JC et al. Epigallocatechin-3-gallate increases maximal oxygen uptake in adult humans. Medicine and Science in Sports and Exercise. 2010.
- Chow HH et al. Pharmacokinetics and safety of green tea polyphenols after multiple-dose administration of epigallocatechin gallate and polyphenon E in healthy individuals. Clinical Cancer Research. 2003.
- Mazzanti G et al. Hepatotoxicity from green tea: a review of the literature and two unpublished cases. European Journal of Clinical Pharmacology. 2009.
- Hu J et al. The safety of green tea and green tea extract consumption in adults. Journal of Nutrition. 2018.
- European Food Safety Authority. Scientific opinion on the safety of green tea catechins. EFSA Journal. 2018.
- Baladia E et al. Effect of green tea or green tea extract consumption on body weight and body composition: systematic review and meta-analysis. Nutricion Hospitalaria. 2014.
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Compounded Medication Notice. Compounded semaglutide and tirzepatide are not FDA-approved. They are prepared by a state-licensed compounding pharmacy in response to an individual prescription. Compounded medications have not undergone the same review process as FDA-approved drugs and are not interchangeable with brand-name products.
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